Publications

Gee, H. Y., …, Beck, B. B., …, Weber, L. T., …, Hildebrandt, F. ARHGDIA mutations cause nephrotic syndrome via defective RHO GTPase signaling. J. Clin. Invest. 123, 3243–3253 (2013).

Beck, B. B., …, Habbig, S., …, Hoppe, B. Novel findings in patients with primary hyperoxaluria type III and implications for advanced molecular testing strategies. Eur. J. Hum. Genet. 21, 162–172 (2013).

Peti-Peterdi, J., Burford, J. L., Hackl, M. J. The First Decade of Using Multiphoton Microscopy for High-Power Kidney Imaging. Am. J. Physiol. Renal Physiol. 302, F227–F233 (2012).

Habbig, S., …, Müller, R.-U., Schwarz, R., Hoehne, M., …, Benzing, T., Schermer, B. The ciliopathy disease protein NPHP9 promotes nuclear delivery and activation of the oncogenic transcriptional regulator TAZ. Hum. Mol. Genet. 21, 5528–5538 (2012).

Habbig, S., Bartram, M. P., Müller, R. U., …, Hoehne, M., …, Benzing, T., Schermer, B. NPHP4, a cilia-associated protein, negatively regulates the Hippo pathway. J. Cell Biol. 193, 633–642 (2011).

Meyer-Schwesinger, C., Dehde, S., Klug, P., Becker, J. U., …, Meyer, T. N. Nephrotic syndrome and subepithelial deposits in a mouse model of immune-mediated anti-podocyte glomerulonephritis. J. Immunol. 187, 3218–3229 (2011).

Meyer-Schwesinger, C., …, Becker, J. U. Bone marrow-derived progenitor cells do not contribute to podocyte turnover in the puromycin aminoglycoside and renal ablation models in rats. Am. J. Pathol. 178, 494–499 (2011).

Agustian, P. A., …, Becker, J. U. Diminished met signaling in podocytes contributes to the development of podocytopenia in transplant glomerulopathy. Am. J. Pathol. 178, 2007–2019 (2011).

Beyer, M., Thabet, Y., Müller, R.-U., …, Schermer, B., …, Schultze, J. L. Repression of the genome organizer SATB1 in regulatory T cells is required for suppressive function and inhibition of effector differentiation. Nat. Immunol. 12, 898–907 (2011).

Worthmann, K., …, Becker, J. U., …, Schiffer, M. Urinary excretion of IGFBP-1 and -3 correlates with disease activity and differentiates focal segmental glomerulosclerosis and minimal change disease. Growth Factors 28, 129–138 (2010).

Schermer, B., Benzing, T. Lipid-Protein Interactions along the Slit Diaphragm of Podocytes. J. Am. Soc. Nephrol. 20, 473–478 (2009).

Becker, J. U., Hoerning, A., Schmid, K. W., Hoyer, P. F. Immigrating progenitor cells contribute to human podocyte turnover. Kidney Int. 72, 1468–1473 (2007).

Landgraf, P., …, Müller, R.-U., Schermer, B., …, Benzing, T., …, Tuschl, T. A mammalian microRNA expression atlas based on small RNA library sequencing. Cell 129, 1401–1414 (2007).

Huber, T. B., Schermer, B., Müller, R. U., Höhne, M., …, Benzing, T. Podocin and MEC-2 bind cholesterol to regulate the activity of associated ion channels. Proc. Natl. Acad. Sci. U.S.A. 103, 17079–17086 (2006).

Beyer, A., …, Ideker, T. Integrated assessment and prediction of transcription factor binding. PLoS Comput. Biol. 2, e70 (2006).

Huber, T. B., Benzing, T. The slit diaphragm: a signaling platform to regulate podocyte function. Curr. Opin. Nephrol. Hypertens. 14, 211–216 (2005).

Benzing, T. Signaling at the Slit Diaphragm. J. Am. Soc. Nephrol. 15, 1382–1391 (2004).